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Beste MOI Dissertationen

Best Doctoral Thesis of the Manchot Graduate School MOI I

Dr. Alida Schäkel

Thesis: AGC-Kinase-Netzwerk in Candida albicans

PI: Prof. Dr. Joachim F. Ernst

Dr. Alida Schäkel's doctoral thesis focused on the topic of ‘AGC kinase network in Candida albicans’. Her research findings contribute to a deeper understanding of the regulatory role of AGC kinases in the pathogenicity mechanisms of Candida.

Dr. Tim Fechner

Thesis: Charakterisierung der neuen, potentiellen Adhäsine Yaa1, Yaa2 und Yaa3 von Chlamydia pneumoniae

PI: Prof. Dr. Johannes H. Hegemann

Dr. Tim Fechtner wrote his doctoral thesis on the topic of ‘Characterization of the new potential adhesins Yaa1, Yaa2, and Yaa3 of Chlamydia pneumoniae’. The results of his research represent another important milestone on the path to developing a vaccine that is not yet available.

Best Doctoral Thesis of the Manchot Graduate School MOI II

Dr. Lasse van Wijlick

Thesis: Adaptation of the human fungal pathogen Candida albicans to host specific environmental conditions mediated by the Ace2 transcription factor
 

PI: Prof. Dr. Joachim Ernst

Best Doctoral Thesis of the Manchot Graduate School MOI III

Dr. Lisa Müller

Thesis: RNA: Regulatory information in human disease and viral infection
 

PI: Prof. Dr. Heiner Schaal

 

Best Doctoral Thesis of the Manchot Graduate School MOI IV

Dr. Fabienne Kocher

Thesis: RNA: Molecular Characterization of Chlamydia – Host Interactions During the Early Stages of Infection
 

PI: Prof. Dr. Johannes Hegemann

Dr. Fabienne Kocher was awarded for her work “Molecular Characterization of Chlamydia–Host Interactions During the Early Stages of Infection.” In her dissertation, Kocher investigated the early stages of infection by the human pathogenic bacteria Chlamydia trachomatis and Chlamydia pneumoniae at the molecular level. She demonstrated how these pathogens, which can replicate only within host cells, use specific surface proteins as well as effector proteins introduced into the host cell at an early stage. In this way, they selectively bind to human epithelial cells and manipulate their cellular structures to facilitate entry into the cell. The results of this research provide new mechanistic insights into key processes of bacterial infection and early host–pathogen interactions.